Critically elucidating the role of selenium Pee webcam chat

Critically elucidating the role of selenium

Selenium levels were decreased in patients with sepsis and in patients with multiple organ failure.Conclusion: Serum concentrations of zinc and PRL are generally low in critically ill children, with a greater decrease in patients with sepsis and in the presence of multiple organ failure.The aim of our study is to detect the serum levels of zinc, selenium, and PRL hormone as important immunomodulators in critically ill children and to investigate the relationship between these immunomodulators and the severity of illness.Subjects and methods: This was a prospective study that included two groups; group 1: 50 critically ill children within 72 hours of intensive care unit admission, and group 2: 30 healthy children as controls.It is associated with redistribution of zinc and selenium to tissues involved in protein synthesis and immune cell proliferation, and this leads to decrease in their serum levels.Zinc is an essential trace element that plays an important role in many biological functions; these include mucosal barrier function, innate and adaptive immunity, oxidative stress responses, and wound healing.PRL acts by stimulating the secretion of other cytokines and the expression of cytokine receptors, which indicates that PRL may influence the immune system.

Prolactin (PRL) is the counterregulatory stress hormone that prevents cortisol/stress-induced lymphocyte apoptosis.The levels of zinc and PRL are inversely correlated with severity of illness.Keywords: critically ill, intensive care, zinc, selenium, prolactin Introduction Critical illness or injury occurs when an impending or existing organ failure impairs the balance of oxygen and substrate supply and demand.Prolactin (PRL) is a protein hormone as well as a cytokine that is synthesized and secreted from specialized cells of the anterior pituitary gland, named lactotrophs.It is also synthesized in many extrapituitary tissues such as cells of the immune system (macrophages, natural killer cells, and T- and B-lymphocytes).

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The assay system utilized one mouse monoclonal anti-PRL antibody for solid-phase (micro titer wells) immobilization and another in the antibody–enzyme (horseradish peroxidase) conjugate solution.

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